Resultados da pesquisa por Autor

1

Overcoming an Energy Crisis?: An Adaptive Role of GSK-3 Inhibition in Ischemia/Reperfusion

Overcoming an Energy Crisis?: An Adaptive Role of GSK-3 Inhibition in Ischemia/Reperfusion

Por Zhai, Peiyong, Sadoshima, Junichi
Publicado em 2008

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

2

Glycogen synthase kinase-3β controls autophagy during myocardial ischemia and reperfusion

Glycogen synthase kinase-3β controls autophagy during myocardial ischemia and reperfusion

Por Zhai, Peiyong, Sadoshima, Junichi
Publicado em 2012

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

3

Oxidative Stress Stimulates Autophagic Flux During Ischemia/Reperfusion

Oxidative Stress Stimulates Autophagic Flux During Ischemia/Reperfusion

Por Hariharan, Nirmala, Zhai, Peiyong, Sadoshima, Junichi
Publicado em 2011

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

4

Nifedipine Inhibits Cardiac Hypertrophy and Left Ventricular Dysfunction in Response to Pressure Overload

Nifedipine Inhibits Cardiac Hypertrophy and Left Ventricular Dysfunction in Response to Pressure Overload

Por Ago, Tetsuro, Yang, Yanfei, Zhai, Peiyong, Sadoshima, Junichi
Publicado em 2010

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

5

Myocardial Injection with GSK-3β-Overexpressing Bone Marrow-Derived Mesenchymal Stem Cells Attenuates Cardiac Dysfunction after Myocardial Infarction

Myocardial Injection with GSK-3β-Overexpressing Bone Marrow-Derived Mesenchymal Stem Cells Attenuates Cardiac Dysfunction after Myocardial Infarction

Por Cho, Jaeyeaon, Zhai, Peiyong, Maejima, Yasuhiro, Sadoshima, Junichi
Publicado em 2011

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

6

Pharmacological modulation of autophagy during cardiac stress

Pharmacological modulation of autophagy during cardiac stress

Por Sciarretta, Sebastiano, Zhai, Peiyong, Volpe, Massimo, Sadoshima, Junichi
Publicado em 2012

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

7

Differential Roles of GSK-3β during Myocardial Ischemia and Ischemia/Reperfusion

Differential Roles of GSK-3β during Myocardial Ischemia and Ischemia/Reperfusion

Por Zhai, Peiyong, Sciarretta, Sebastiano, Galeotti, Jonathan, Volpe, Massimo, Sadoshima, Junichi
Publicado em 2011

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

8

Thioredoxin 1 Negatively Regulates Angiotensin II-Induced Cardiac Hypertrophy through Upregulation of miR-98/let-7

Thioredoxin 1 Negatively Regulates Angiotensin II-Induced Cardiac Hypertrophy through Upregulation of miR-98/let-7

Por Yang, Yanfei, Ago, Tetsuro, Zhai, Peiyong, Abdellatif, Maha, Sadoshima, Junichi
Publicado em 2010

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

9

Constitutively active MEK1 rescues cardiac dysfunction caused by overexpressed GSK-3α during aging and hemodynamic pressure overload

Constitutively active MEK1 rescues cardiac dysfunction caused by overexpressed GSK-3α during aging and hemodynamic pressure overload

Por Maejima, Yasuhiro, Galeotti, Jonathan, Molkentin, Jeffery D., Sadoshima, Junichi, Zhai, Peiyong
Publicado em 2012

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

10

Nicotinamide Mononucleotide, an Intermediate of NAD(+) Synthesis, Protects the Heart from Ischemia and Reperfusion

Nicotinamide Mononucleotide, an Intermediate of NAD(+) Synthesis, Protects the Heart from Ischemia and Reperfusion

Por Yamamoto, Takanobu, Byun, Jaemin, Zhai, Peiyong, Ikeda, Yoshiyuki, Oka, Shinichi, Sadoshima, Junichi
Publicado em 2014

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

11

Mst1-mediated phosphorylation of Bcl-xL is required for myocardial reperfusion injury

Mst1-mediated phosphorylation of Bcl-xL is required for myocardial reperfusion injury

Por Nakamura, Michinari, Zhai, Peiyong, Del Re, Dominic P., Maejima, Yasuhiro, Sadoshima, Junichi
Publicado no JCI Insight (2016)

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

12

NADPH oxidase 4 (Nox4) is a major source of oxidative stress in the failing heart

NADPH oxidase 4 (Nox4) is a major source of oxidative stress in the failing heart

Por Kuroda, Junya, Ago, Tetsuro, Matsushima, Shouji, Zhai, Peiyong, Schneider, Michael D., Sadoshima, Junichi
Publicado em 2010

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

13

Elucidation of Thioredoxin Target Protein Networks in Mouse

Elucidation of Thioredoxin Target Protein Networks in Mouse

Por Fu, Cexiong, Wu, Changgong, Liu, Tong, Ago, Tetsuro, Zhai, Peiyong, Sadoshima, Junichi, Li, Hong
Publicado em 2009

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

14

Suppression of ERR targets by a PPARα/Sirt1 complex in the failing heart

Suppression of ERR targets by a PPARα/Sirt1 complex in the failing heart

Por Oka, Shin-ichi, Zhai, Peiyong, Alcendor, Ralph, Park, Ji Yeon, Tian, Bin, Sadoshima, Junichi
Publicado em 2012

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

15

Rheb is a critical regulator of autophagy during myocardial ischemia: pathophysiological implications in obesity and metabolic syndrome

Rheb is a critical regulator of autophagy during myocardial ischemia: pathophysiological implications in obesity and metabolic syndrome

Por Sciarretta, Sebastiano, Zhai, Peiyong, Shao, Dan, Maejima, Yasuhiro, Robbins, Jeffrey, Volpe, Massimo, Condorelli, Gianluigi, Sadoshima, Junichi
Publicado em 2012

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

16

Activation of Nox4 in the Endoplasmic Reticulum Promotes Cardiomyocyte Autophagy and Survival during Energy Stress through the PERK/eIF-2α/ATF4 pathway

Activation of Nox4 in the Endoplasmic Reticulum Promotes Cardiomyocyte Autophagy and Survival during Energy Stress through the PERK/eIF-2α/ATF4 pathway

Por Sciarretta, Sebastiano, Zhai, Peiyong, Shao, Dan, Zablocki, Daniela, Nagarajan, Narayani, Terada, Lance S., Volpe, Massimo, Sadoshima, Junichi
Publicado em 2013

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

17

PPARα Association With Sirt1 Suppresses Cardiac Fatty Acid Metabolism in the Failing Heart

PPARα Association With Sirt1 Suppresses Cardiac Fatty Acid Metabolism in the Failing Heart

Por Oka, Shin-ichi, Zhai, Peiyong, Yamamoto, Takanobu, Ikeda, Yoshiyuki, Byun, Jaemin, Hsu, Chiao-Po, Sadoshima, Junichi
Publicado no Circ Heart Fail (2015)

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

18

A Redox-Dependent Mechanism for Regulation of AMPK Activation by Thioredoxin1 during Energy Starvation

A Redox-Dependent Mechanism for Regulation of AMPK Activation by Thioredoxin1 during Energy Starvation

Por Shao, Dan, Oka, Shin-ichi, Liu, Tong, Zhai, Peiyong, Ago, Tetsuro, Sciarretta, Sebastiano, Li, Hong, Sadoshima, Junichi
Publicado em 2014

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

19

Increased Oxidative Stress in the Nucleus Caused by Nox4 Mediates Oxidation of HDAC4 and Cardiac Hypertrophy

Increased Oxidative Stress in the Nucleus Caused by Nox4 Mediates Oxidation of HDAC4 and Cardiac Hypertrophy

Por Matsushima, Shouji, Kuroda, Junya, Ago, Tetsuro, Zhai, Peiyong, Park, Ji Yeon, Xie, Lai-Hua, Tian, Bin, Sadoshima, Junichi
Publicado em 2012

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

20

MuRF1 Negatively Regulates Pathological Cardiac Hypertrophy Through Downregulation of Calcineurin A

MuRF1 Negatively Regulates Pathological Cardiac Hypertrophy Through Downregulation of Calcineurin A

Por Maejima, Yasuhiro, Usui, Soichiro, Zhai, Peiyong, Takamura, Masayuki, Kaneko, Shuichi, Zablocki, Daniela, Yokota, Mitsuhiro, Isobe, Mitsuaki, Sadoshima, Junichi
Publicado em 2014

Obter o texto integral
Obter o texto integral
Obter o texto integral

Artigo

1
2
3
Seguinte
[3]