HDAC Inhibition Counteracts Metastatic Re-Activation of Prostate Cancer Cells Induced by Chronic mTOR Suppression

This study was designed to investigate whether epigenetic modulation by histone deacetylase (HDAC) inhibition might circumvent resistance towards the mechanistic target of rapamycin (mTOR) inhibitor temsirolimus in a prostate cancer cell model. Parental (par) and temsirolimus-resistant (res) PC3 pro...

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Bibliografiset tiedot
Julkaisussa:Cells
Päätekijät: Makarević, Jasmina, Rutz, Jochen, Juengel, Eva, Maxeiner, Sebastian, Mani, Jens, Vallo, Stefan, Tsaur, Igor, Roos, Frederik, Chun, Felix K.-H., Blaheta, Roman A.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: MDPI 2018
Aiheet:
Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6162415/
https://ncbi.nlm.nih.gov/pubmed/30200497
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/cells7090129
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