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Mice thrive without Cdk4 and Cdk2
Mammalian cell division is thought to be driven by sequential activation of several Cyclin‐dependent kinases (Cdk), mainly Cdk4, Cdk6, Cdk2 and Cdk1. Since mice lacking Cdk4, Cdk6 or Cdk2 are viable, it has been proposed that they play compensatory roles. We report here that mice lacking Cdk4 and Cd...
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| 出版年: | Mol Oncol |
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| 主要な著者: | , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
John Wiley and Sons Inc.
2007
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5543859/ https://ncbi.nlm.nih.gov/pubmed/19383288 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molonc.2007.03.001 |
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