Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI

During recovery by regeneration after AKI, proximal tubule cells can fail to redifferentiate, undergo premature growth arrest, and become atrophic. The atrophic tubules display pathologically persistent signaling increases that trigger production of profibrotic peptides, proliferation of interstitia...

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Vydáno v:J Am Soc Nephrol
Hlavní autoři: Lan, Rongpei, Geng, Hui, Singha, Prajjal K., Saikumar, Pothana, Bottinger, Erwin P., Weinberg, Joel M., Venkatachalam, Manjeri A.
Médium: Artigo
Jazyk:Inglês
Vydáno: American Society of Nephrology 2016
Témata:
Basic Research
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5084876/
https://ncbi.nlm.nih.gov/pubmed/27000065
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2015020177
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