Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI

During recovery by regeneration after AKI, proximal tubule cells can fail to redifferentiate, undergo premature growth arrest, and become atrophic. The atrophic tubules display pathologically persistent signaling increases that trigger production of profibrotic peptides, proliferation of interstitia...

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Bibliografski detalji
Izdano u:J Am Soc Nephrol
Glavni autori: Lan, Rongpei, Geng, Hui, Singha, Prajjal K., Saikumar, Pothana, Bottinger, Erwin P., Weinberg, Joel M., Venkatachalam, Manjeri A.
Format: Artigo
Jezik:Inglês
Izdano: American Society of Nephrology 2016
Teme:
Basic Research
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5084876/
https://ncbi.nlm.nih.gov/pubmed/27000065
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2015020177
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