Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI

During recovery by regeneration after AKI, proximal tubule cells can fail to redifferentiate, undergo premature growth arrest, and become atrophic. The atrophic tubules display pathologically persistent signaling increases that trigger production of profibrotic peptides, proliferation of interstitia...

詳細記述

保存先:
書誌詳細
出版年:J Am Soc Nephrol
主要な著者: Lan, Rongpei, Geng, Hui, Singha, Prajjal K., Saikumar, Pothana, Bottinger, Erwin P., Weinberg, Joel M., Venkatachalam, Manjeri A.
フォーマット: Artigo
言語:Inglês
出版事項: American Society of Nephrology 2016
主題:
Basic Research
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC5084876/
https://ncbi.nlm.nih.gov/pubmed/27000065
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2015020177
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