Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI

During recovery by regeneration after AKI, proximal tubule cells can fail to redifferentiate, undergo premature growth arrest, and become atrophic. The atrophic tubules display pathologically persistent signaling increases that trigger production of profibrotic peptides, proliferation of interstitia...

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Detalhes bibliográficos
Publicado no:J Am Soc Nephrol
Main Authors: Lan, Rongpei, Geng, Hui, Singha, Prajjal K., Saikumar, Pothana, Bottinger, Erwin P., Weinberg, Joel M., Venkatachalam, Manjeri A.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society of Nephrology 2016
Assuntos:
Basic Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5084876/
https://ncbi.nlm.nih.gov/pubmed/27000065
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2015020177
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