Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI

During recovery by regeneration after AKI, proximal tubule cells can fail to redifferentiate, undergo premature growth arrest, and become atrophic. The atrophic tubules display pathologically persistent signaling increases that trigger production of profibrotic peptides, proliferation of interstitia...

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Dades bibliogràfiques
Publicat a:J Am Soc Nephrol
Autors principals: Lan, Rongpei, Geng, Hui, Singha, Prajjal K., Saikumar, Pothana, Bottinger, Erwin P., Weinberg, Joel M., Venkatachalam, Manjeri A.
Format: Artigo
Idioma:Inglês
Publicat: American Society of Nephrology 2016
Matèries:
Basic Research
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC5084876/
https://ncbi.nlm.nih.gov/pubmed/27000065
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2015020177
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