טוען...
Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI
During recovery by regeneration after AKI, proximal tubule cells can fail to redifferentiate, undergo premature growth arrest, and become atrophic. The atrophic tubules display pathologically persistent signaling increases that trigger production of profibrotic peptides, proliferation of interstitia...
שמור ב:
| הוצא לאור ב: | J Am Soc Nephrol |
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| Main Authors: | , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society of Nephrology
2016
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5084876/ https://ncbi.nlm.nih.gov/pubmed/27000065 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2015020177 |
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