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AlkB homolog 2 (ABH2) mediated repair of ethenoadenine lesions in mammalian DNA

Endogenous formation of the mutagenic DNA adduct 1,N(6)-ethenoadenosine (εA) originates from lipid peroxidation. Elevated levels of εA in cancer-prone tissues suggest a role for this adduct in the development of some cancers. The base excision repair (BER) pathway has been considered the principal r...

詳細記述

保存先:
書誌詳細
出版年:Cancer Res
主要な著者: Ringvoll, Jeanette, Moen, Marivi N., Nordstrand, Line M., Meira, Lisiane B., Pang, Bo, Bekkelund, Anders, Dedon, Peter C., Bjelland, Svein, Samson, Leona D., Falnes, Pål Ø., Klungland, Arne
フォーマット: Artigo
言語:Inglês
出版事項: 2008
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC4725713/
https://ncbi.nlm.nih.gov/pubmed/18519673
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/0008-5472.CAN-08-0796
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