ΔF508 CFTR Pool in the Endoplasmic Reticulum Is Increased by Calnexin Overexpression

The most common cystic fibrosis transmembrane conductance regulator (CFTR) mutant in cystic fibrosis patients, ΔF508 CFTR, is retained in the endoplasmic reticulum (ER) and is consequently degraded by the ubiquitin-proteasome pathway known as ER-associated degradation (ERAD). Because the prolonged i...

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Detaylı Bibliyografya
Asıl Yazarlar: Okiyoneda, Tsukasa, Harada, Kazutsune, Takeya, Motohiro, Yamahira, Kaori, Wada, Ikuo, Shuto, Tsuyoshi, Suico, Mary Ann, Hashimoto, Yasuaki, Kai, Hirofumi
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: The American Society for Cell Biology 2004
Konular:
Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC329241/
https://ncbi.nlm.nih.gov/pubmed/14595111
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E03-06-0379
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