ΔF508 CFTR Pool in the Endoplasmic Reticulum Is Increased by Calnexin Overexpression

The most common cystic fibrosis transmembrane conductance regulator (CFTR) mutant in cystic fibrosis patients, ΔF508 CFTR, is retained in the endoplasmic reticulum (ER) and is consequently degraded by the ubiquitin-proteasome pathway known as ER-associated degradation (ERAD). Because the prolonged i...

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Dettagli Bibliografici

Pubblicato in:	Mol Biol Cell
Autori principali:	Okiyoneda, Tsukasa, Harada, Kazutsune, Takeya, Motohiro, Yamahira, Kaori, Wada, Ikuo, Shuto, Tsuyoshi, Suico, Mary Ann, Hashimoto, Yasuaki, Kai, Hirofumi
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	American Society for Cell Biology 2004
Soggetti:	Articles
Accesso online:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC329241/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/14595111/ https://ncbi.nlm.nih.govhttps://doi.org/10.1091/mbc.E03-06-0379
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