ΔF508 CFTR Pool in the Endoplasmic Reticulum Is Increased by Calnexin Overexpression

The most common cystic fibrosis transmembrane conductance regulator (CFTR) mutant in cystic fibrosis patients, ΔF508 CFTR, is retained in the endoplasmic reticulum (ER) and is consequently degraded by the ubiquitin-proteasome pathway known as ER-associated degradation (ERAD). Because the prolonged i...

Celý popis

Podrobná bibliografie

Vydáno v:	Mol Biol Cell
Hlavní autoři:	Okiyoneda, Tsukasa, Harada, Kazutsune, Takeya, Motohiro, Yamahira, Kaori, Wada, Ikuo, Shuto, Tsuyoshi, Suico, Mary Ann, Hashimoto, Yasuaki, Kai, Hirofumi
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	American Society for Cell Biology 2004
Témata:	Articles
On-line přístup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC329241/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/14595111/ https://ncbi.nlm.nih.govhttps://doi.org/10.1091/mbc.E03-06-0379
Tagy:	Přidat tag Žádné tagy, Buďte první, kdo vytvoří štítek k tomuto záznamu!