ΔF508 CFTR Pool in the Endoplasmic Reticulum Is Increased by Calnexin Overexpression

The most common cystic fibrosis transmembrane conductance regulator (CFTR) mutant in cystic fibrosis patients, ΔF508 CFTR, is retained in the endoplasmic reticulum (ER) and is consequently degraded by the ubiquitin-proteasome pathway known as ER-associated degradation (ERAD). Because the prolonged i...

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書目詳細資料

發表在:	Mol Biol Cell
Principais autores:	Okiyoneda, Tsukasa, Harada, Kazutsune, Takeya, Motohiro, Yamahira, Kaori, Wada, Ikuo, Shuto, Tsuyoshi, Suico, Mary Ann, Hashimoto, Yasuaki, Kai, Hirofumi
格式:	Artigo
語言:	Inglês
出版:	American Society for Cell Biology 2004
主題:	Articles
在線閱讀:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC329241/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/14595111/ https://ncbi.nlm.nih.govhttps://doi.org/10.1091/mbc.E03-06-0379
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