ΔF508 CFTR Pool in the Endoplasmic Reticulum Is Increased by Calnexin Overexpression

The most common cystic fibrosis transmembrane conductance regulator (CFTR) mutant in cystic fibrosis patients, ΔF508 CFTR, is retained in the endoplasmic reticulum (ER) and is consequently degraded by the ubiquitin-proteasome pathway known as ER-associated degradation (ERAD). Because the prolonged i...

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Bibliografski detalji
Glavni autori: Okiyoneda, Tsukasa, Harada, Kazutsune, Takeya, Motohiro, Yamahira, Kaori, Wada, Ikuo, Shuto, Tsuyoshi, Suico, Mary Ann, Hashimoto, Yasuaki, Kai, Hirofumi
Format: Artigo
Jezik:Inglês
Izdano: The American Society for Cell Biology 2004
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Articles
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC329241/
https://ncbi.nlm.nih.gov/pubmed/14595111
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E03-06-0379
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