ΔF508 CFTR Pool in the Endoplasmic Reticulum Is Increased by Calnexin Overexpression

The most common cystic fibrosis transmembrane conductance regulator (CFTR) mutant in cystic fibrosis patients, ΔF508 CFTR, is retained in the endoplasmic reticulum (ER) and is consequently degraded by the ubiquitin-proteasome pathway known as ER-associated degradation (ERAD). Because the prolonged i...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
मुख्य लेखकों: Okiyoneda, Tsukasa, Harada, Kazutsune, Takeya, Motohiro, Yamahira, Kaori, Wada, Ikuo, Shuto, Tsuyoshi, Suico, Mary Ann, Hashimoto, Yasuaki, Kai, Hirofumi
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: The American Society for Cell Biology 2004
विषय:
Articles
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC329241/
https://ncbi.nlm.nih.gov/pubmed/14595111
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E03-06-0379
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