ΔF508 CFTR Pool in the Endoplasmic Reticulum Is Increased by Calnexin Overexpression

The most common cystic fibrosis transmembrane conductance regulator (CFTR) mutant in cystic fibrosis patients, ΔF508 CFTR, is retained in the endoplasmic reticulum (ER) and is consequently degraded by the ubiquitin-proteasome pathway known as ER-associated degradation (ERAD). Because the prolonged i...

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Main Authors: Okiyoneda, Tsukasa, Harada, Kazutsune, Takeya, Motohiro, Yamahira, Kaori, Wada, Ikuo, Shuto, Tsuyoshi, Suico, Mary Ann, Hashimoto, Yasuaki, Kai, Hirofumi
Formato: Artigo
Idioma:Inglês
Publicado em: The American Society for Cell Biology 2004
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC329241/
https://ncbi.nlm.nih.gov/pubmed/14595111
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E03-06-0379
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