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ΔF508 CFTR Pool in the Endoplasmic Reticulum Is Increased by Calnexin Overexpression
The most common cystic fibrosis transmembrane conductance regulator (CFTR) mutant in cystic fibrosis patients, ΔF508 CFTR, is retained in the endoplasmic reticulum (ER) and is consequently degraded by the ubiquitin-proteasome pathway known as ER-associated degradation (ERAD). Because the prolonged i...
Αποθηκεύτηκε σε:
| Κύριοι συγγραφείς: | , , , , , , , , |
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| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
The American Society for Cell Biology
2004
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC329241/ https://ncbi.nlm.nih.gov/pubmed/14595111 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E03-06-0379 |
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