Schedule-dependent interaction between anticancer treatments

Por Chen, Sheng-hong, Forrester, William, Lahav, Galit
Publicado no Science (2016)

Nuclear matrix attachment regions antagonize methylation-dependent repression of long-range enhancer–promoter interactions

Por Forrester, William C., Fernández, Luis A., Grosschedl, Rudolf
Publicado em 1999

Cell-to-cell variation in p53 dynamics leads to fractional killing

Por Paek, Andrew L., Liu, Julia C., Loewer, Alexander, Forrester, William C., Lahav, Galit
Publicado no Cell (2016)

A DNA vector-based RNAi technology to suppress gene expression in mammalian cells

Por Sui, Guangchao, Soohoo, Christina, Affar, El Bachir, Gay, Frédérique, Shi, Yujiang, Forrester, William C., Shi, Yang
Publicado em 2002

p53 pulses lead to distinct patterns of gene expression albeit similar DNA binding dynamics

Por Hafner, Antonina, Stewart-Ornstein, Jacob, Purvis, Jeremy E., Forrester, William C., Bulyk, Martha L., Lahav, Galit
Publicado no Nat Struct Mol Biol (2017)

Recurrent ubiquitin B silencing in gynecological cancers establishes dependence on ubiquitin C

Por Kedves, Alexia T., Gleim, Scott, Liang, Xiaoyou, Bonal, Dennis M., Sigoillot, Frederic, Harbinski, Fred, Sanghavi, Sneha, Benander, Christina, George, Elizabeth, Gokhale, Prafulla C., Nguyen, Quang-De, Kirschmeier, Paul T., Distel, Robert J., Jenkins, Jeremy, Goldberg, Michael S., Forrester, William C.
Publicado no J Clin Invest (2017)

Structure of the DDB1-CRBN E3 ubiquitin ligase in complex with thalidomide

Por Fischer, Eric S., Böhm, Kerstin, Lydeard, John R., Yang, Haidi, Stadler, Michael B., Cavadini, Simone, Nagel, Jane, Serluca, Fabrizio, Acker, Vincent, Lingaraju, Gondichatnahalli M., Tichkule, Ritesh B., Schebesta, Michael, Forrester, William C., Schirle, Markus, Hassiepen, Ulrich, Ottl, Johannes, Hild, Marc, Beckwith, Rohan E. J., Harper, J. Wade, Jenkins, Jeremy L., Thomä, Nicolas H.
Publicado no Nature (2014)