Resultados da pesquisa - brain ((((tumor 1) or (tumor rna))s) or (((tumor 1) or (tumor rna)))) treat

Pesquisas alternativas:
tumor rna » tumor renal (Expandir a Pesquisa)
tumor 1 » tumor _ (Expandir a Pesquisa), tumor a (Expandir a Pesquisa)

1

Blockade of SDF-1 after irradiation inhibits tumor recurrences of autochthonous brain tumors in rats

Blockade of SDF-1 after irradiation inhibits tumor recurrences of autochthonous brain tumors in rats

Por Liu, Shie-Chau, Alomran, Reem, Chernikova, Sophia B., Lartey, Fred, Stafford, Jason, Jang, Taichang, Merchant, Milton, Zboralski, Dirk, Zöllner, Stefan, Kruschinski, Anna, Klussmann, Sven, Recht, Lawrence, Brown, J. Martin
Publicado em 2014

“...-nitrosourea. RESULTS: Rats treated in utero with N-ethyl-N-nitrosourea began to die of brain tumors from...”
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2

NT-16 NANOPARTICLE-MEDIATED DELIVERY OF ANTI-Ape1 siRNA SENSITIZES PEDIATRIC BRAIN TUMOR CELLS TO RADIATION THERAPY BY INHIBITING DNA REPAIR

NT-16 NANOPARTICLE-MEDIATED DELIVERY OF ANTI-Ape1 siRNA SENSITIZES PEDIATRIC BRAIN TUMOR CELLS TO RADIATION THERAPY BY INHIBITING DNA REPAIR

Por Kievit, Forrest, Stephen, Zachary, Wang, Kui, Dayringer, Christopher, Ellenbogen, Richard, Silber, John, Zhang, Miqin
Publicado em 2014

“... implicated in conferring radiation resistance in pediatric brain tumors. However, inhibiting Ape1 activity...”
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3

DDIS-05. DEVELOPMENT OF AN INHIBITOR AGAINST MUSASHI-1 AND ITS USE IN BRAIN TUMOR THERAPY

DDIS-05. DEVELOPMENT OF AN INHIBITOR AGAINST MUSASHI-1 AND ITS USE IN BRAIN TUMOR THERAPY

Por Yi, Caihong
Publicado no Neuro Oncol (2017)

“...We identified the RNA binding protein Musashi1 (Msi1) as a major driver of glioblastoma...”
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4

STEM-28. DOT1L EPIGENETICALLY REGULATES GBM BRAIN TUMOR STEM CELLS

STEM-28. DOT1L EPIGENETICALLY REGULATES GBM BRAIN TUMOR STEM CELLS

Por Bozek, Danielle, Wang, Alice, Hao, Xiaoguang, Johnston, Michael, Luchman, H Artee, Weiss, Samuel
Publicado no Neuro Oncol (2017)

“..., of mice treated with EPZ-5676, revealed that DOT1L inhibition promotes neuronal and astrocytic...”
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5

SCDT-11. ASTROCYTIC S1P3 SIGNALING MODULATES BLOOD-TUMOR BARRIER PERMEABILITY IN BRAIN METASTASES

SCDT-11. ASTROCYTIC S1P3 SIGNALING MODULATES BLOOD-TUMOR BARRIER PERMEABILITY IN BRAIN METASTASES

Por Gril, Brunilde, Paranjape, Anurag N, Woditschka, Stephan, Hanson, Jeffrey, Wu, Xiaolin, Duchnowska, Renata, Brastianos, Priscilla K, Peer, Cody, Figg, William D, Pauly, Gary T, Schneider, Joel P, Steeg, Patricia S
Publicado no Neuro Oncol (2017)

“... colonize the brain, the BBB evolves into a blood-tumor barrier (BTB) and remains a potent barrier...”
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6

TMIC-19. ASTROCYTIC S1P3 SIGNALING MODULATES BLOOD-TUMOR BARRIER PERMEABILITY IN BRAIN METASTASES

TMIC-19. ASTROCYTIC S1P3 SIGNALING MODULATES BLOOD-TUMOR BARRIER PERMEABILITY IN BRAIN METASTASES

Por Gril, Brunilde, Paranjape, Anurag N, Woditschka, Stephan, Hanson, Jeffrey, Wu, Xiaolin, Duchnowska, Renata, Brastianos, Priscilla K, Peer, Cody, Figg, William D, Pauly, Gary T, Schneider, Joel P, Steeg, Patricia
Publicado no Neuro Oncol (2017)

“... colonize the brain, the BBB evolves into a blood-tumor barrier (BTB) and remains a potent barrier...”
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7

Targeting Polo-like kinase 1 in SMARCB1 deleted atypical teratoid rhabdoid tumor

Targeting Polo-like kinase 1 in SMARCB1 deleted atypical teratoid rhabdoid tumor

Por Alimova, Irina, Pierce, Angela M., Harris, Peter, Donson, Andrew, Birks, Diane K., Prince, Eric, Balakrishnan, Ilango, Foreman, Nicholas K., Kool, Marcel, Hoffman, Lindsey, Venkataraman, Sujatha, Vibhakar, Rajeev
Publicado no Oncotarget (2017)

“...Atypical teratoid rhabdoid tumor (ATRT) is an aggressive and malignant pediatric brain tumor. Polo...”
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8

TMIC-12. TUMOR-HOMING RNA-NANOPARTICLES REPROGRAM IMMUNE CELLS IN THE BRAIN TUMOR MICROENVIRONMENT

TMIC-12. TUMOR-HOMING RNA-NANOPARTICLES REPROGRAM IMMUNE CELLS IN THE BRAIN TUMOR MICROENVIRONMENT

Por Grippin, Adam, Mendez-Gomez, Hector, Wummer, Brandon, Wildes, Tyler, Dyson, Kyle, Valladares, Daniel, Dobson, Jon, Sayour, Elias, Mitchell, Duane
Publicado no Neuro Oncol (2018)

“...BACKGROUND: Brain tumors are particularly difficult to treat due to their relative isolation behind...”
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9

Decreased cyclin B1 expression contributes to G2 delay in human brain tumor cells after treatment with camptothecin.

Decreased cyclin B1 expression contributes to G2 delay in human brain tumor cells after treatment with camptothecin.

Por Janss, A. J., Maity, A., Tang, C. B., Muschel, R. J., McKenna, W. G., Sutton, L., Phillips, P. C.
Publicado em 2001

“... is a common response of brain tumor cells to chemotherapy with topoisomerase I inhibitors and that a...”
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10

Over-expressed lncRNA HOTAIRM1 promotes tumor growth and invasion through up-regulating HOXA1 and sequestering G9a/EZH2/Dnmts away from the HOXA1 gene in glioblastoma multiforme

Over-expressed lncRNA HOTAIRM1 promotes tumor growth and invasion through up-regulating HOXA1...

Por Li, Qi, Dong, Chengya, Cui, Jiayue, Wang, Yubo, Hong, Xinyu
Publicado no J Exp Clin Cancer Res (2018)

“...BACKGROUND: Glioblastoma multiforme (GBM) is the common primary brain tumor classified the most...”
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11

Inhibition of GLO1 in Glioblastoma Multiforme Increases DNA-AGEs, Stimulates RAGE Expression, and Inhibits Brain Tumor Growth in Orthotopic Mouse Models

Inhibition of GLO1 in Glioblastoma Multiforme Increases DNA-AGEs, Stimulates RAGE Expression, and Inhibits Brain Tumor Growth in Orthotopic Mouse Models

Por Jandial, Rahul, Neman, Josh, Lim, Punnajit P., Tamae, Daniel, Kowolik, Claudia M., Wuenschell, Gerald E., Shuck, Sarah C., Ciminera, Alexandra K., De Jesus, Luis R., Ouyang, Ching, Chen, Mike Y., Termini, John
Publicado no Int J Mol Sci (2018)

“... multiforme (GBM), the most aggressive and difficult to treat malignancy of the brain. Elevated GLO1...”
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12

Inhibition of GLO1 in Glioblastoma Multiforme Increases DNA-AGEs, Stimulates RAGE Expression, and Inhibits Brain Tumor Growth in Orthotopic Mouse Models

Inhibition of GLO1 in Glioblastoma Multiforme Increases DNA-AGEs, Stimulates RAGE Expression, and Inhibits Brain Tumor Growth in Orthotopic Mouse Models

Por Rahul Jandial, Josh Neman, Punnajit P. Lim, Daniel Tamae, Claudia M. Kowolik, Gerald E. Wuenschell, Sarah C. Shuck, Alexandra K. Ciminera, Luis R. De Jesus, Ching Ouyang, Mike Y. Chen, John Termini
Publicado em 2018-01-01

Assuntos: “...glyoxalase 1...”
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13

Tight junction protein claudin-1 is differentially expressed in craniopharyngioma subtypes and indicates invasive tumor growth

Tight junction protein claudin-1 is differentially expressed in craniopharyngioma subtypes and indicates invasive tumor growth

Por Stache, Christina, Hölsken, Annett, Fahlbusch, Rudolf, Flitsch, Jörg, Schlaffer, Sven-Martin, Buchfelder, Michael, Buslei, Rolf
Publicado em 2014

“.... Accordingly, siRNA treatment for CLDN1 altered tumor cell migration in vitro. CONCLUSION: CLDN1 represents a...”
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14

Expression of NR1I3 in mouse lung tumors induced by the tobacco-specific nitrosamine 4-(methylnitrosamino)-4-(3-pyridyl)-1-butanone

Expression of NR1I3 in mouse lung tumors induced by the tobacco-specific nitrosamine 4-(methylnitrosamino)-4-(3-pyridyl)-1-butanone

Por Fukumasu, H., Cordeiro, Y.G., Rochetti, A.L., Barra, C.N., Sámora, T.S., Strefezzi, R.F., Dagli, M.L.Z.
Publicado no Braz J Med Biol Res (2015)

“.... Gene expression analysis also showed an inverse correlation between NR1I3 mRNA expression and tumor...”
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15

NF-κB upregulation through epigenetic silencing of LDOC1 drives tumor biology and specific immunophenotype in Group A ependymoma

NF-κB upregulation through epigenetic silencing of LDOC1 drives tumor biology and specific immunophenotype in Group A ependymoma

Por Griesinger, Andrea M, Witt, Davis A, Grob, Sydney T, Georgio Westover, Sabrina R, Donson, Andrew M, Sanford, Bridget, Mulcahy Levy, Jean M, Wong, Randall, Moreira, Daniel C, DeSisto, John A, Balakrishnan, Ilango, Hoffman, Lindsey M, Handler, Michael H, Jones, Kenneth L, Vibhakar, Rajeev, Venkataraman, Sujatha, Foreman, Nicholas K
Publicado no Neuro Oncol (2017)

“... expression in GpA compared with GpB EPN, and in other pediatric brain tumors. EPN cells treated with 5AZA-DC...”
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16

SPARC overexpression alters microRNA expression profiles involved in tumor progression

SPARC overexpression alters microRNA expression profiles involved in tumor progression

Por Ahir, Bhavesh K., Elias, Nasya M., Lakka, Sajani S.
Publicado no Genes Cancer (2017)

“...Medulloblastoma is the most common malignant brain tumor in children. SPARC (secreted protein...”
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17

Immunotherapy of intracranial G422 glioblastoma with dendritic cells pulsed with tumor extract or RNA

Immunotherapy of intracranial G422 glioblastoma with dendritic cells pulsed with tumor extract or RNA

Por Zhang, Zhe, Tang, Ling-ling, Zhan, Ren-ya, Tong, Ying, Yao, Hang-ping, Du, Li-an
Publicado em 2004

“... with G422 tumor cells. All the treated mice were followed for survival. Some mice brains were removed...”
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18

Gene- and viral-based therapies for brain tumors

Gene- and viral-based therapies for brain tumors

Por Asadi-Moghaddam, Kaveh, Chiocca, E. Antonio
Publicado em 2009

“... genetic material to fight or prevent disease with brain tumors being among one of the first human...”
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19

Disulfiram modulates stemness and metabolism of brain tumor initiating cells in atypical teratoid/rhabdoid tumors

Disulfiram modulates stemness and metabolism of brain tumor initiating cells in atypical teratoid/rhabdoid tumors

Por Choi, Seung Ah, Choi, Jung Won, Wang, Kyu-Chang, Phi, Ji Hoon, Lee, Ji Yeoun, Park, Kyung Duk, Eum, Dayoung, Park, Sung-Hye, Kim, Il Han, Kim, Seung-Ki
Publicado no Neuro Oncol (2015)

“...BACKGROUND: Atypical teratoid/rhabdoid tumors (AT/RT) are among the most malignant pediatric brain...”
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20

Major Histocompatibility Complex (MHC) Class I Gene Expression in Single Neurons of the Central Nervous System: Differential Regulation by Interferon (IFN)-γ and Tumor Necrosis Factor (TNF)-α

Major Histocompatibility Complex (MHC) Class I Gene Expression in Single Neurons of the Central...

Por Neumann, H., Schmidt, H., Cavalié, A., Jenne, D., Wekerle, H.
Publicado em 1997

“... necrosis factor-α (TNF-α) on the induction of MHC class I–related genes in functionally mature brain...”
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