טוען...
Genetic inhibition of RIPK1 reduces cell death and improves functional outcome after intracerebral hemorrhage in mice
BACKGROUND AND PURPOSE: Recent studies using cultured cells and rodent intracerebral hemorrhage (ICH) models have implicated receptor interacting protein kinase-1 (RIPK1) as a driver of programmed necrosis and secondary injury based on use of chemical inhibitors. However, these inhibitors have off t...
שמור ב:
| הוצא לאור ב: | Stroke |
|---|---|
| Main Authors: | , , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2017
|
| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6366458/ https://ncbi.nlm.nih.gov/pubmed/28765287 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/STROKEAHA.117.017702 |
| תגים: |
הוספת תג
אין תגיות, היה/י הראשונ/ה לתייג את הרשומה!
|