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Genetic inhibition of RIPK1 reduces cell death and improves functional outcome after intracerebral hemorrhage in mice
BACKGROUND AND PURPOSE: Recent studies using cultured cells and rodent intracerebral hemorrhage (ICH) models have implicated receptor interacting protein kinase-1 (RIPK1) as a driver of programmed necrosis and secondary injury based on use of chemical inhibitors. However, these inhibitors have off t...
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| Publicat a: | Stroke |
|---|---|
| Autors principals: | , , , , , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2017
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6366458/ https://ncbi.nlm.nih.gov/pubmed/28765287 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/STROKEAHA.117.017702 |
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