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Stabilization of α-Synuclein Fibril Clusters Prevents Fragmentation and Reduces Seeding Activity and Toxicity

Protein misfolding results in the accumulation of aggregated β-sheet-rich structures in Parkinson’s disease (PD) and Alzheimer’s disease (AD). The toxic oligomer hypothesis stipulates that prefibrillar assemblies, such as soluble oligomers or protofibrils, are responsible for the poor prognosis of t...

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Detalles Bibliográficos
Publicado en:Biochemistry
Main Authors: Lam, Huy T., Graber, Michael C., Gentry, Katherine A., Bieschke, Jan
Formato: Artigo
Idioma:Inglês
Publicado: 2016
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC5706464/
https://ncbi.nlm.nih.gov/pubmed/26799377
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1021/acs.biochem.5b01168
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