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Stabilization of α-Synuclein Fibril Clusters Prevents Fragmentation and Reduces Seeding Activity and Toxicity

Protein misfolding results in the accumulation of aggregated β-sheet-rich structures in Parkinson’s disease (PD) and Alzheimer’s disease (AD). The toxic oligomer hypothesis stipulates that prefibrillar assemblies, such as soluble oligomers or protofibrils, are responsible for the poor prognosis of t...

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Bibliographic Details
Published in:Biochemistry
Main Authors: Lam, Huy T., Graber, Michael C., Gentry, Katherine A., Bieschke, Jan
Format: Artigo
Language:Inglês
Published: 2016
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Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC5706464/
https://ncbi.nlm.nih.gov/pubmed/26799377
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1021/acs.biochem.5b01168
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