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α-Synuclein binds TOM20 and inhibits mitochondrial protein import in Parkinson’s disease
α-Synuclein accumulation and mitochondrial dysfunction have both been strongly implicated in the pathogenesis of Parkinson’s disease (PD), and the two appear to be related. Mitochondrial dysfunction leads to accumulation and oligomerization of α-synuclein, and increased levels of α-synuclein cause m...
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| 出版年: | Sci Transl Med |
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| 主要な著者: | , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5016095/ https://ncbi.nlm.nih.gov/pubmed/27280685 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1126/scitranslmed.aaf3634 |
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