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DNA end joining becomes less efficient and more error-prone during cellular senescence
Accumulation of somatic mutations is thought to contribute to the aging process. Genomic instability has been shown to increase during aging, suggesting an aberrant function of DNA double-strand break (DSB) repair. Surprisingly, DSB repair has not been examined with respect to cellular senescence. T...
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| Päätekijät: | , , , , |
|---|---|
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
National Academy of Sciences
2004
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC419656/ https://ncbi.nlm.nih.gov/pubmed/15123826 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0400726101 |
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