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Glioblastoma Resistance to Anti-VEGF Therapy: Has the Challenge Been MET?
In glioblastoma cells the receptor tyrosine kinase c-Met is upregulated in response to Bevacizumab and plays an important role in promoting invasion and tumor recurrence. These data support novel links between VEGF-A and HGF and suggest that c-Met and its signaling effectors may be effective targets...
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| Autor principal: | |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2013
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| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3618531/ https://ncbi.nlm.nih.gov/pubmed/23403631 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/1078-0432.CCR-13-0051 |
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