Activation of Apoptosis Signaling Eliminates CD34(+) Progenitor Cells in Blast Crisis CML Independent of Response to Tyrosine Kinase Inhibitors

Схожие документы

• Triptolide induces cell death independent of cellular responses to imatinib in blast crisis CML cells including quiescent CD34(+) primitive progenitor cells
  по: Mak, Duncan H., et al.
  Опубликовано: (2009)
• Synergistic effects of p53 activation via MDM2 inhibition in combination with inhibition of Bcl-2 or Bcr-Abl in CD34(+) proliferating and quiescent chronic myeloid leukemia blast crisis cells
  по: Carter, Bing Z., et al.
  Опубликовано: (2015)
• The elusive CML stem cell: does it matter and how do we eliminate it?
  по: Carter, Bing Z., et al.
  Опубликовано: (2010)
• Combined inhibition of β-catenin and Bcr–Abl synergistically targets tyrosine kinase inhibitor-resistant blast crisis chronic myeloid leukemia blasts and progenitors in vitro and in vivo
  по: Zhou, H, et al.
  Опубликовано: (2017)
• Combined Targeting of BCL-2 and BCR-ABL Tyrosine Kinase Eradicates Chronic Myeloid Leukemia Stem Cells
  по: Carter, Bing Z., et al.
  Опубликовано: (2016)