Deficiency of terminal complement pathway inhibitor promotes neuronal tau pathology and degeneration in mice

BACKGROUND: The neuronal microtubule-associated protein tau becomes hyperphosphorylated and forms aggregates in tauopathies but the processes leading to this pathological hallmark are not understood. Because tauopathies are accompanied by neuroinflammation and the complement cascade forms a key inna...

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Detaylı Bibliyografya
Asıl Yazarlar: Britschgi, Markus, Takeda-Uchimura, Yoshiko, Rockenstein, Edward, Johns, Hudson, Masliah, Eliezer, Wyss-Coray, Tony
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: BioMed Central 2012
Konular:
Short Report
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3511294/
https://ncbi.nlm.nih.gov/pubmed/22989354
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1742-2094-9-220
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