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Deficiency of terminal complement pathway inhibitor promotes neuronal tau pathology and degeneration in mice
BACKGROUND: The neuronal microtubule-associated protein tau becomes hyperphosphorylated and forms aggregates in tauopathies but the processes leading to this pathological hallmark are not understood. Because tauopathies are accompanied by neuroinflammation and the complement cascade forms a key inna...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
BioMed Central
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3511294/ https://ncbi.nlm.nih.gov/pubmed/22989354 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1742-2094-9-220 |
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