Glucagon Protects Against Impaired NMDA-Mediated Cerebrovasodilation and Cerebral Autoregulation during Hypotension after Brain Injury by Activating cAMP Protein Kinase A and Inhibiting Upregulation of tPA

Outcome of traumatic brain injury (TBI) is impaired by hyperglycemia, hypotension, and glutamate, and improved by insulin. Insulin reduces glutamate concentration, making it uncertain whether its beneficial effect accrues from euglycemia. Glucagon decreases CNS glutamate, lessens neuronal cell injur...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
Main Authors: Armstead, William M., Kiessling, J. Willis, Cines, Douglas B., Higazi, Abd Al-Roof
פורמט: Artigo
שפה:Inglês
יצא לאור: Mary Ann Liebert, Inc. 2011
נושאים:
Original Articles
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC3057207/
https://ncbi.nlm.nih.gov/pubmed/21375400
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/neu.2010.1659
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