ATM regulates Mre11-dependent DNA end-degradation and microhomology-mediated end joining

The human disorder ataxia telangiectasia (AT), which is characterized by genetic instability and neurodegeneration, results from mutation of the ataxia telangiectasia mutated (ATM) kinase. The loss of ATM leads to cell cycle checkpoint deficiencies and other DNA damage signaling defects that do not...

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Detaylı Bibliyografya
Asıl Yazarlar: Rahal, Elias A, Henricksen, Leigh A, Li, Yuling, Williams, R Scott, Tainer, John A, Dixon, Kathleen
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Landes Bioscience 2010
Konular:
Report
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3040963/
https://ncbi.nlm.nih.gov/pubmed/20647759
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/cc.9.14.12363
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