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Cerebral cavernous malformations proteins inhibit Rho kinase to stabilize vascular integrity
Endothelial cell–cell junctions regulate vascular permeability, vasculogenesis, and angiogenesis. Familial cerebral cavernous malformations (CCMs) in humans result from mutations of CCM2 (malcavernin, OSM, MGC4607), PDCD10 (CCM3), or KRIT1 (CCM1), a Rap1 effector which stabilizes endothelial cell–ce...
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| Hoofdauteurs: | , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
The Rockefeller University Press
2010
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2856024/ https://ncbi.nlm.nih.gov/pubmed/20308363 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20091258 |
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