Neprilysin Overexpression Inhibits Plaque Formation But Fails to Reduce Pathogenic Aβ Oligomers and Associated Cognitive Deficits in Human Amyloid Precursor Protein Transgenic Mice

समान संसाधन

• Ablation of Cellular Prion Protein Does Not Ameliorate Abnormal Neural Network Activity or Cognitive Dysfunction in the J20 Line of Human Amyloid Precursor Protein Transgenic Mice
  द्वारा: Cissé, Moustapha, और अन्य
  प्रकाशित: (2011)
• Reduction in Mitochondrial Superoxide Dismutase Modulates Alzheimer's Disease-Like Pathology and Accelerates the Onset of Behavioral Changes in Human Amyloid Precursor Protein Transgenic Mice
  द्वारा: Esposito, Luke, और अन्य
  प्रकाशित: (2006)
• Enkephalin Elevations Contribute to Neuronal and Behavioral Impairments in a Transgenic Mouse Model of Alzheimer's Disease
  द्वारा: Meilandt, William J., और अन्य
  प्रकाशित: (2008)
• Elimination of the Class A Scavenger Receptor Does Not Affect Amyloid Plaque Formation or Neurodegeneration in Transgenic Mice Expressing Human Amyloid Protein Precursors
  द्वारा: Huang, Frederick, और अन्य
  प्रकाशित: (1999)
• Amyloid Plaques and Amyloid-β Oligomers: An Ongoing Debate
  द्वारा: Lesné, Sylvain, और अन्य
  प्रकाशित: (2005)