Carboxyl-terminal-truncated apolipoprotein E4 causes Alzheimer's disease-like neurodegeneration and behavioral deficits in transgenic mice

Lignende værker

• Neuron-Specific Apolipoprotein E4 Proteolysis Is Associated with Increased Tau Phosphorylation in Brains of Transgenic Mice
  af: Brecht, Walter J., et al.
  Udgivet: (2004)
• Expression of Human Apolipoprotein E3 or E4 in the Brains ofApoe(−/−) Mice: Isoform-Specific Effects on Neurodegeneration
  af: Buttini, Manuel, et al.
  Udgivet: (1999)
• Bioactive TGF-β can associate with lipoproteins and is enriched in those containing apolipoprotein E3
  af: Tesseur, Ina, et al.
  Udgivet: (2009)
• Deficiency in neuronal TGF-β signaling promotes neurodegeneration and Alzheimer’s pathology
  af: Tesseur, Ina, et al.
  Udgivet: (2006)
• Cellular Source of Apolipoprotein E4 Determines Neuronal Susceptibility to Excitotoxic Injury in Transgenic Mice
  af: Buttini, Manuel, et al.
  Udgivet: (2010)