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Mice Null for Sox18 Are Viable and Display a Mild Coat Defect
We have previously shown that Sox18 is expressed in developing vascular endothelium and hair follicles during mouse embryogenesis and that point mutations in Sox18 are the underlying cause of cardiovascular and hair follicle defects in ragged (Ra) mice. Here we describe the analysis of Sox18(−/−) mi...
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主要な著者: | , , , , |
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フォーマット: | Artigo |
言語: | Inglês |
出版事項: |
American Society for Microbiology
2000
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主題: | |
オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC102189/ https://ncbi.nlm.nih.gov/pubmed/11094083 |
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