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Mice Null for Sox18 Are Viable and Display a Mild Coat Defect

We have previously shown that Sox18 is expressed in developing vascular endothelium and hair follicles during mouse embryogenesis and that point mutations in Sox18 are the underlying cause of cardiovascular and hair follicle defects in ragged (Ra) mice. Here we describe the analysis of Sox18(−/−) mi...

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書誌詳細
主要な著者: Pennisi, David, Bowles, Josephine, Nagy, Andras, Muscat, George, Koopman, Peter
フォーマット: Artigo
言語:Inglês
出版事項: American Society for Microbiology 2000
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC102189/
https://ncbi.nlm.nih.gov/pubmed/11094083
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