Mice Null for Sox18 Are Viable and Display a Mild Coat Defect
We have previously shown that Sox18 is expressed in developing vascular endothelium and hair follicles during mouse embryogenesis and that point mutations in Sox18 are the underlying cause of cardiovascular and hair follicle defects in ragged (Ra) mice. Here we describe the analysis of Sox18(−/−) mi...
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| Pubblicato in: | Mol Cell Biol |
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| Autori principali: | , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Taylor & Francis
2000
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC102189/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11094083/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.20.24.9331-9336.2000 |
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