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Mice Null for Sox18 Are Viable and Display a Mild Coat Defect

We have previously shown that Sox18 is expressed in developing vascular endothelium and hair follicles during mouse embryogenesis and that point mutations in Sox18 are the underlying cause of cardiovascular and hair follicle defects in ragged (Ra) mice. Here we describe the analysis of Sox18(−/−) mi...

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Detalhes bibliográficos
Main Authors: Pennisi, David, Bowles, Josephine, Nagy, Andras, Muscat, George, Koopman, Peter
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Microbiology 2000
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC102189/
https://ncbi.nlm.nih.gov/pubmed/11094083
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