Mice Null for Sox18 Are Viable and Display a Mild Coat Defect

We have previously shown that Sox18 is expressed in developing vascular endothelium and hair follicles during mouse embryogenesis and that point mutations in Sox18 are the underlying cause of cardiovascular and hair follicle defects in ragged (Ra) mice. Here we describe the analysis of Sox18(−/−) mi...

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Dades bibliogràfiques

Publicat a:	Mol Cell Biol
Autors principals:	Pennisi, David, Bowles, Josephine, Nagy, Andras, Muscat, George, Koopman, Peter
Format:	Artigo
Idioma:	Inglês
Publicat:	Taylor & Francis 2000
Matèries:	Mammalian Genetic Models with Minimal or Complex Phenotypes
Accés en línia:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC102189/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11094083/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.20.24.9331-9336.2000
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