Mice Null for Sox18 Are Viable and Display a Mild Coat Defect

We have previously shown that Sox18 is expressed in developing vascular endothelium and hair follicles during mouse embryogenesis and that point mutations in Sox18 are the underlying cause of cardiovascular and hair follicle defects in ragged (Ra) mice. Here we describe the analysis of Sox18(−/−) mi...

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Détails bibliographiques

Publié dans:	Mol Cell Biol
Auteurs principaux:	Pennisi, David, Bowles, Josephine, Nagy, Andras, Muscat, George, Koopman, Peter
Format:	Artigo
Langue:	Inglês
Publié:	Taylor & Francis 2000
Sujets:	Mammalian Genetic Models with Minimal or Complex Phenotypes
Accès en ligne:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC102189/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11094083/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.20.24.9331-9336.2000
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