Old drugs learn new tricks

Por Strittmatter, Stephen M
Publicado no Nat Med (2014)

Emerging Mechanisms in Alzheimer’s Disease and Their Therapeutic Implications

Por Strittmatter, Stephen M.
Publicado no Biol Psychiatry (2018)

P2Y1 purinergic receptors in sensory neurons: contribution to touch-induced impulse generation.

Por Nakamura, F, Strittmatter, S M
Publicado em 1996

The reticulons: a family of proteins with diverse functions

Por Yang, Yvonne S, Strittmatter, Stephen M
Publicado em 2007

The N-Terminal Domain of Nogo-A Inhibits Cell Adhesion and Axonal Outgrowth by an Integrin-Specific Mechanism

Por Hu, Fenghua, Strittmatter, Stephen M.
Publicado em 2008

Oligomers of Amyloid β Prevent Physiological Activation of the Cellular Prion Protein-Metabotropic Glutamate Receptor 5 Complex by Glutamate in Alzheimer Disease

Por Haas, Laura T., Strittmatter, Stephen M.
Publicado no J Biol Chem (2016)

Can regenerating axons recapitulate developmental guidance during recovery from spinal cord injury?

Por Harel, Noam Y., Strittmatter, Stephen M.
Publicado em 2006

Functional MRI and other non-invasive imaging technologies: Providing visual biomarkers for spinal cord structure and function after injury

Por Harel, Noam Y., Strittmatter, Stephen M.
Publicado em 2008

Cellular Prion Protein Mediates the Toxicity of β-Amyloid Oligomers: Implications for Alzheimer Disease

Por Nygaard, Haakon B., Strittmatter, Stephen M.
Publicado em 2009

β-amyloid oligomers and cellular prion protein in Alzheimer's disease

Por Gunther, Erik C., Strittmatter, Stephen M.
Publicado em 2009

Nogo Receptor Interacts with Brain APP and Aβ to Reduce Pathologic Changes in Alzheimer’s Transgenic Mice

Por Park, James H., Strittmatter, Stephen M.
Publicado em 2007

Axon Regeneration in the Peripheral and Central Nervous Systems

Por Huebner, Eric A., Strittmatter, Stephen M.
Publicado em 2009

The CRMP Family of Proteins and Their Role in Sema3A Signaling

Por Schmidt, Eric F., Strittmatter, Stephen M.
Publicado em 2007

Lynx for Braking Plasticity

Por Higley, Michael J., Strittmatter, Stephen M.
Publicado em 2010

Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer disease

Por Um, Ji Won, Strittmatter, Stephen M.
Publicado em 2013

Nogo limits neural plasticity and recovery from injury

Por Schwab, Martin E., Strittmatter, Stephen M.
Publicado em 2014

Cellular Prion Protein as a Receptor for Amyloid-β Oligomers in Alzheimer’s Disease

Por Salazar, Santiago V., Strittmatter, Stephen M.
Publicado no Biochem Biophys Res Commun (2016)

Binding Sites for Amyloid-β Oligomers and Synaptic Toxicity

Por Smith, Levi M., Strittmatter, Stephen M.
Publicado no Cold Spring Harb Perspect Med (2017)

Synaptotoxic Signaling by Amyloid Beta Oligomers in Alzheimer's Disease Through Prion Protein and mGluR5

Por Brody, A.H., Strittmatter, Stephen M.
Publicado no Adv Pharmacol (2017)

Physical separation and characterization of two types of benzodiazepine receptors.

Por Lo, M M, Strittmatter, S M, Snyder, S H
Publicado em 1982