Histone methylation in Huntington’s disease: are bivalent promoters the critical targets?

Por Zsindely, Nóra, Bodai, László
Publicado no Neural Regen Res (2018)

A novel target for Huntington’s disease: ERK at the crossroads of signaling: The ERK signaling pathway is implicated in Huntington’s disease and its upregulation ameliorates pathol...

Por Bodai, László, Marsh, J. Lawrence
Publicado em 2012

Pcaf Modulates Polyglutamine Pathology in a Drosophila Model of Huntington's Disease

Por Bodai, Laszlo, Pallos, Judit, Thompson, Leslie Michels, Marsh, J. Lawrence
Publicado em 2012

Systematic genetic interaction studies identify histone demethylase Utx as potential target for ameliorating Huntington’s disease

Por Song, Wan, Zsindely, Nóra, Faragó, Anikó, Marsh, J Lawrence, Bodai, László
Publicado no Hum Mol Genet (2018)

Ecdysone Induced Gene Expression Is Associated with Acetylation of Histone H3 Lysine 23 in Drosophila melanogaster

Por Bodai, László, Zsindely, Nóra, Gáspár, Renáta, Kristó, Ildikó, Komonyi, Orbán, Boros, Imre Miklós
Publicado em 2012

Functional characterization and gene expression profiling of Drosophila melanogaster short dADA2b isoform-containing dSAGA complexes

Por Pankotai, Tibor, Zsindely, Nóra, Vamos, Edith E, Komonyi, Orbán, Bodai, László, Boros, Imre M
Publicado em 2013

ERK activation by the polyphenols fisetin and resveratrol provides neuroprotection in multiple models of Huntington's disease

Por Maher, Pamela, Dargusch, Richard, Bodai, Laszlo, Gerard, Paul E., Purcell, Judith M., Marsh, J. Lawrence
Publicado em 2011

Inhibition of specific HDACs and sirtuins suppresses pathogenesis in a Drosophila model of Huntington’s disease

Por Pallos, Judit, Bodai, Laszlo, Lukacsovich, Tamas, Purcell, Judith M., Steffan, Joan S., Thompson, Leslie Michels, Marsh, J. Lawrence
Publicado em 2008

The loss of histone H3 lysine 9 acetylation due to dSAGA-specific dAda2b mutation influences the expression of only a small subset of genes

Por Zsindely, Nóra, Pankotai, Tibor, Újfaludi, Zsuzsanna, Lakatos, Dániel, Komonyi, Orbán, Bodai, László, Tora, László, Boros, Imre M.
Publicado em 2009

The Homologous Drosophila Transcriptional Adaptors ADA2a and ADA2b Are both Required for Normal Development but Have Different Functions

Por Pankotai, Tibor, Komonyi, Orbán, Bodai, László, Újfaludi, Zsuzsanna, Muratoglu, Selen, Ciurciu, Anita, Tora, László, Szabad, János, Boros, Imre
Publicado em 2005

Identification of combinatorial drug regimens for treatment of Huntington's disease using Drosophila

Por Agrawal, Namita, Pallos, Judit, Slepko, Natalia, Apostol, Barbara L., Bodai, Laszlo, Chang, Ling-Wen, Chiang, Ann-Shyn, Thompson, Leslie Michels, Marsh, J. Lawrence
Publicado em 2005

A cell-based assay for aggregation inhibitors as therapeutics of polyglutamine-repeat disease and validation in Drosophila

Por Apostol, Barbara L., Kazantsev, Alexsey, Raffioni, Simona, Illes, Katalin, Pallos, Judit, Bodai, Laszlo, Slepko, Natalia, Bear, James E., Gertler, Frank B., Hersch, Steven, Housman, David E., Marsh, J. Lawrence, Thompson, Leslie Michels
Publicado em 2003

Targeting H3K4 trimethylation in Huntington disease

Por Vashishtha, Malini, Ng, Christopher W., Yildirim, Ferah, Gipson, Theresa A., Kratter, Ian H., Bodai, Laszlo, Song, Wan, Lau, Alice, Labadorf, Adam, Vogel-Ciernia, Annie, Troncosco, Juan, Ross, Christopher A., Bates, Gillian P., Krainc, Dimitri, Sadri-Vakili, Ghazaleh, Finkbeiner, Steven, Marsh, J. Lawrence, Housman, David E., Fraenkel, Ernest, Thompson, Leslie M.
Publicado em 2013