Elevation of Mitochondrial Transmembrane Potential and Reactive Oxygen Intermediate Levels Are Early Events and Occur Independently from Activation of Caspases in Fas Signaling

Por Banki, Katalin, Hutter, Eliza, Gonchoroff, Nick J., Perl, Andras
Publicado em 1999

Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity

Por Perl, Andras, Gergely, Peter, Nagy, Gyorgy, Koncz, Agnes, Banki, Katalin
Publicado em 2004

Activation of the Mechanistic Target of Rapamycin in SLE: Explosion of Evidence in the Last Five Years

Por Oaks, Zachary, Winans, Thomas, Huang, Nick, Banki, Katalin, Perl, Andras
Publicado no Curr Rheumatol Rep (2016)

Study of transaldolase deficiency in urine samples by capillary LC-MS/MS

Por Vas, György, Conkrite, Karina, Amidon, Wendy, Qian, Yueming, Bánki, Katalin, Perl, András
Publicado em 2006

Mitochondrial Dysfunction in the Liver and Antiphospholipid Antibody Production Precede Disease Onset and Respond to Rapamycin in Lupus‐Prone Mice

Por Oaks, Zachary, Winans, Thomas, Caza, Tiffany, Fernandez, David, Liu, Yuxin, Landas, Steve K., Banki, Katalin, Perl, Andras
Publicado no Arthritis Rheumatol (2016)

HRES-1/Rab4 Promotes the Formation of LC3(+) Autophagosomes and the Accumulation of Mitochondria during Autophagy

Por Talaber, Gergely, Miklossy, Gabriella, Oaks, Zachary, Liu, Yuxin, Tooze, Sharon A., Chudakov, Dmitriy M., Banki, Katalin, Perl, Andras
Publicado em 2014

Mitochondrial Hyperpolarization and ATP Depletion in Patients With Systemic Lupus Erythematosus

Por Gergely, Peter, Grossman, Craig, Niland, Brian, Puskas, Ferenc, Neupane, Hom, Allam, Fatme, Banki, Katalin, Phillips, Paul E., Perl, Andras
Publicado em 2002

Cleavage of Transaldolase by Granzyme B Causes the Loss of Enzymatic Activity with Retention of Antigenicity for Multiple Sclerosis Patients

Por Niland, Brian, Miklossy, Gabriella, Banki, Katalin, Biddison, William E., Casciola-Rosen, Livia, Rosen, Antony, Martinvalet, Denis, Lieberman, Judy, Perl, Andras
Publicado em 2010

Liver injury correlates with biomarkers of autoimmunity and disease activity and represents an organ system involvement in patients with systemic lupus erythematosus

Por Liu, Yuxin, Yu, Jianghong, Oaks, Zachary, Marchena-Mendez, Ivan, Francis, Lisa, Bonilla, Eduardo, Aleksiejuk, Phillip, Patel, Jessica, Banki, Katalin, Landas, Steve K., Perl, Andras
Publicado no Clin Immunol (2015)

Deletion of Ser-171 causes inactivation, proteasome-mediated degradation and complete deficiency of human transaldolase

Por Grossman, Craig E., Niland, Brian, Stancato, Christina, Verhoeven, Nanda M., van der Knaap, Marjo S., Jakobs, Cornelis, Brown, Lawrence M., Vajda, Sandor, Banki, Katalin, Perl, Andras
Publicado em 2004

Transaldolase is essential for maintenance of the mitochondrial transmembrane potential and fertility of spermatozoa

Por Perl, Andras, Qian, Yueming, Chohan, Kazim R., Shirley, Cynthia R., Amidon, Wendy, Banerjee, Sanjay, Middleton, Frank A., Conkrite, Karina L., Barcza, Maureen, Gonchoroff, Nick, Suarez, Susan S., Banki, Katalin
Publicado em 2006

Prevention of hepatocarcinogenesis and increased susceptibility to acetaminophen-induced liver failure in transaldolase-deficient mice by N-acetylcysteine

Por Hanczko, Robert, Fernandez, David R., Doherty, Edward, Qian, Yueming, Vas, Gyorgy, Niland, Brian, Telarico, Tiffany, Garba, Adinoyi, Banerjee, Sanjay, Middleton, Frank A., Barrett, Donna, Barcza, Maureen, Banki, Katalin, Landas, Steve K., Perl, Andras
Publicado em 2009

HRES-1/Rab4-mediated depletion of Drp1 impairs mitochondrial homeostasis and represents a target for treatment in SLE

Por Caza, Tiffany N, Fernandez, David R, Talaber, Gergely, Oaks, Zachary, Haas, Mark, Madaio, Michael P, Lai, Zhi-wei, Miklossy, Gabriella, Singh, Ram R, Chudakov, Dmitriy M, Malorni, Walter, Middleton, Frank, Banki, Katalin, Perl, Andras
Publicado em 2014