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Complement activity is regulated in C3 glomerulopathy by IgG–factor H fusion proteins with and without properdin targeting domains
C3 glomerulopathy is characterized by accumulation of complement C3 within glomeruli. Causes include, but are not limited to, abnormalities in factor H, the major negative regulator of the complement alternative pathway. Factor H-deficient (Cfh(-/-)) mice develop C3 glomerulopathy together with a re...
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| Izdano u: | Kidney Int |
|---|---|
| Glavni autori: | , , , , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
Elsevier
2021
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7863913/ https://ncbi.nlm.nih.gov/pubmed/33129896 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.kint.2020.09.028 |
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