USP22 functions as an oncogenic driver in prostate cancer by regulating cell proliferation and DNA repair

Emerging evidence indicates the deubiquitinase USP22 regulates transcriptional activation and modification of target substrates to promote pro-oncogenic phenotypes. Here, in vivo characterization of tumor-associated USP22 upregulation and unbiased interrogation of USP22-regulated functions in vitro...

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Bibliografiska uppgifter
I publikationen:Cancer Res
Huvudupphovsmän: McCann, Jennifer J., Vasilevskaya, Irina A., Neupane, Neermala Poudel, Shafi, Ayesha A., McNair, Christopher, Dylgjeri, Emanuela, Mandigo, Amy C., Schiewer, Matthew J., Schrecengost, Randy S., Gallagher, Peter, Stanek, Timothy J., McMahon, Steven B., Berman-Booty, Lisa D., Ostrander, William F., Knudsen, Karen E.
Materialtyp: Artigo
Språk:Inglês
Publicerad: 2019
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Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC7814394/
https://ncbi.nlm.nih.gov/pubmed/31740444
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/0008-5472.CAN-19-1033
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