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FAK regulates IL-33 expression by controlling chromatin accessibility at c-Jun motifs
Focal adhesion kinase (FAK) localizes to focal adhesions and is overexpressed in many cancers. FAK can also translocate to the nucleus, where it binds to, and regulates, several transcription factors, including MBD2, p53 and IL-33, to control gene expression by unknown mechanisms. We have used ATAC-...
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| 出版年: | Sci Rep |
|---|---|
| 主要な著者: | , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Nature Publishing Group UK
2021
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7794255/ https://ncbi.nlm.nih.gov/pubmed/33420223 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-020-80111-9 |
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