A novel gene-diet interaction promotes organismal lifespan and host protection during infection via the mitochondrial UPR

Cells use a variety of mechanisms to maintain optimal mitochondrial function including the mitochondrial unfolded protein response (UPR(mt)). The UPR(mt) mitigates mitochondrial dysfunction by differentially regulating mitoprotective gene expression through the transcription factor ATFS-1. Since UPR...

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Detaylı Bibliyografya
Yayımlandı:PLoS Genet
Asıl Yazarlar: Amin, Mustafi Raisa, Mahmud, Siraje Arif, Dowgielewicz, Jonathan L., Sapkota, Madhab, Pellegrino, Mark W.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Public Library of Science 2020
Konular:
Research Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC7781476/
https://ncbi.nlm.nih.gov/pubmed/33338044
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pgen.1009234
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