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Oxidative stress mediates thalidomide-induced pain by targeting peripheral TRPA1 and central TRPV4

BACKGROUND: The mechanism underlying the pain symptoms associated with chemotherapeutic-induced peripheral neuropathy (CIPN) is poorly understood. Transient receptor potential ankyrin 1 (TRPA1), TRP vanilloid 4 (TRPV4), TRPV1, and oxidative stress have been implicated in several rodent models of CIP...

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Τόπος έκδοσης:	BMC Biol
Κύριοι συγγραφείς:	De Logu, Francesco, Trevisan, Gabriela, Marone, Ilaria Maddalena, Coppi, Elisabetta, Padilha Dalenogare, Diéssica, Titiz, Mustafa, Marini, Matilde, Landini, Lorenzo, Souza Monteiro de Araujo, Daniel, Li Puma, Simone, Materazzi, Serena, De Siena, Gaetano, Geppetti, Pierangelo, Nassini, Romina
Μορφή:	Artigo
Γλώσσα:	Inglês
Έκδοση:	BioMed Central 2020
Θέματα:	Research Article
Διαθέσιμο Online:	https://ncbi.nlm.nih.gov/pmc/articles/PMC7737339/ https://ncbi.nlm.nih.gov/pubmed/33317522 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12915-020-00935-9
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Oxidative stress mediates thalidomide-induced pain by targeting peripheral TRPA1 and central TRPV4

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