Tafazzin Mutation Affecting Cardiolipin Leads to Increased Mitochondrial Superoxide Anions and Mitophagy Inhibition in Barth Syndrome

Lignende værker

• Cardiolipin remodeling by TAZ/tafazzin is selectively required for the initiation of mitophagy
  af: Hsu, Paul, et al.
  Udgivet: (2015)
• The Cardiolipin Transacylase, Tafazzin, Associates with Two Distinct Respiratory Components Providing Insight into Barth Syndrome
  af: Claypool, Steven M., et al.
  Udgivet: (2008)
• TAZ encodes tafazzin, a transacylase essential for cardiolipin formation and central to the etiology of Barth syndrome
  af: Garlid, Anders O., et al.
  Udgivet: (2019)
• Correction for The Cardiolipin Transacylase, Tafazzin, Associates with Two Distinct Respiratory Components Providing Insight into Barth Syndrome
  Udgivet: (2012)
• Deletion of the Cardiolipin-specific Phospholipase Cld1 Rescues Growth and Life Span Defects in the Tafazzin Mutant: IMPLICATIONS FOR BARTH SYNDROME
  af: Ye, Cunqi, et al.
  Udgivet: (2014)