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Dysregulation of Mitochondrial Ca(2+) Uptake and Sarcolemma Repair Underlie Muscle Weakness and Wasting in Patients and Mice Lacking MICU1
Muscle function is regulated by Ca(2+), which mediates excitation-contraction coupling, energy metabolism, adaptation to exercise, and sarcolemmal repair. Several of these actions rely on Ca(2+) delivery to the mitochondrial matrix via the mitochondrial Ca(2+) uniporter, the pore of which is formed...
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| Опубликовано в: : | Cell Rep |
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| Главные авторы: | , , , , , , , , , |
| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
2019
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7007691/ https://ncbi.nlm.nih.gov/pubmed/31665639 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.celrep.2019.09.063 |
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