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Dysregulation of Mitochondrial Ca(2+) Uptake and Sarcolemma Repair Underlie Muscle Weakness and Wasting in Patients and Mice Lacking MICU1
Muscle function is regulated by Ca(2+), which mediates excitation-contraction coupling, energy metabolism, adaptation to exercise, and sarcolemmal repair. Several of these actions rely on Ca(2+) delivery to the mitochondrial matrix via the mitochondrial Ca(2+) uniporter, the pore of which is formed...
Guardat en:
| Publicat a: | Cell Rep |
|---|---|
| Autors principals: | , , , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2019
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7007691/ https://ncbi.nlm.nih.gov/pubmed/31665639 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.celrep.2019.09.063 |
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