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mTORC1 directly inhibits AMPK to promote cell proliferation under nutrient stress
Central to cellular metabolism and cell proliferation are highly conserved signalling pathways controlled by mammalian target of rapamycin (mTOR) and AMP-activated protein kinase (AMPK)(1,2), dysregulation of which are implicated in pathogenesis of major human diseases such as cancer and type 2 diab...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | Nat Metab |
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| Κύριοι συγγραφείς: | , , , , , , , , , , , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
2020
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6986917/ https://ncbi.nlm.nih.gov/pubmed/31993556 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s42255-019-0157-1 |
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