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Defective postreplication repair of UV photoproducts in melanoma: a mutator phenotype
In this issue, the Gabrielli laboratory and collaborators address the bulky CPD lesions created in DNA when UV joins two adjacent pyrimidines (thymine or cytosine), leading to skin cancers such as melanoma (Pavey S et al. (2019) Mol Oncol). Our understanding of postreplication repair mechanisms for...
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| Vydáno v: | Mol Oncol |
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| Hlavní autoři: | , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
John Wiley and Sons Inc.
2019
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6944110/ https://ncbi.nlm.nih.gov/pubmed/31821728 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/1878-0261.12612 |
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