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VEGF signalling enhances lesion burden in KRIT1 deficient mice
The exact molecular mechanisms underlying CCM pathogenesis remain a complicated and controversial topic. Our previous work illustrated an important VEGF signalling loop in KRIT1 depleted endothelial cells. As VEGF is a major mediator of many vascular pathologies, we asked whether the increased VEGF...
Tallennettuna:
| Julkaisussa: | J Cell Mol Med |
|---|---|
| Päätekijät: | , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
John Wiley and Sons Inc.
2019
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6933401/ https://ncbi.nlm.nih.gov/pubmed/31746130 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.14773 |
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