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VEGF signalling enhances lesion burden in KRIT1 deficient mice
The exact molecular mechanisms underlying CCM pathogenesis remain a complicated and controversial topic. Our previous work illustrated an important VEGF signalling loop in KRIT1 depleted endothelial cells. As VEGF is a major mediator of many vascular pathologies, we asked whether the increased VEGF...
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| 發表在: | J Cell Mol Med |
|---|---|
| Main Authors: | , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
John Wiley and Sons Inc.
2019
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6933401/ https://ncbi.nlm.nih.gov/pubmed/31746130 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.14773 |
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